What is it and how to treat it
Coccidiosis in Chickens
Coccidiosis causes the poultry industry to suffer a considerable economic loss, especially in the production of broiler chickens. Chickens are
susceptible to at least 11 species of coccidia. The most common species in Alberta are Eimeria tenella, which causes the cecal or bloody type of
coccidiosis, and E. acervulina and E. maxima, which cause chronic intestinal coccidiosis.
The broiler industry in Alberta produces about 35 million birds each year. To control coccidiosis, yearly expenditures on anticoccidial drugs total
approximately $650,000. For poultry producers, this is probably the largest expenditure for medication.
General life cycle of coccidia
Stages of coccidia in chickens appear both within the host as well as outside. The developmental stages in the chicken give rise to a
microscopic egg (called an oocyst) which is passed out in the droppings. Under proper conditions of temperature and moisture the oocyst
develops within one to two days to form a sporulated oocyst which is capable of infecting other chickens. At this stage the ooycst contains eight
bodies (called sporozoites), each of which is capable of entering a cell in the chicken's intestine after the oocyst is eaten. When sporozoites
enter the cells, they divide many times producing either a few or many offspring (merozoites). The numbers produced depend on the species of
coccidia involved. Each merozoite in turn may enter another intestinal cell. This cycle may be repeated several times. Because of this cyclic
multiplication, large numbers of intestinal cells are destroyed. Eventually, the cycle stops and sex cells (male and female) are produced. The
male fertilizes the female to produce and oocyst which ruptures from the intestinal cell and passes in the droppings. Thousands of oocysts may
be passed in the droppings of an infected chicken. Therefore, poultry raised in crowded or unsanitary conditions are at great risk of becoming
Special features of the life cycle
Eimeria tenella This parasite develops in the cells of the cecca which are the two blind sacs near the end of the intestine. It is one of
the most pathogenic (disease producing) coccidia to infect chickens.
E. necatrix - E. necatrix develops in the small intestine (early stages) and later in the cecum (sexual stages). Like E. tenella, it
develops within deeper tissues of the small intestine and is a major pathogen of poultry.
E. accervulina and E. maxima - Both species develop in epithelial cells, primarily in the upper part of the small intestine.
How do birds become infected?
Normally, most birds pass small numbers of oocysts in their droppings without apparent ill effects. Coccidiosis becomes important as a disease
when animals live, or are reared, under conditions that permit the build-up of infective oocysts in the environment. The intensive rearing of
domestic chickens may provide these conditions.
Young chickens pick up the infection from contaminated premises (soil, houses, utensils, etc.). These may have been contaminated previously
by other young infected birds or by adult birds that have recovered from the condition. Wet areas around water fountains are a source of
infection. Oocysts remain viable in litter for many months. In this way they can contaminate a farm from year to year. Oocysts are killed by
freezing, extreme dryness and high temperatures.
How do coccidia harm chickens?
Several factors influence the severity of infection. Some of these are:
- The number of oocysts eaten. Generally, an increase in the number of oocysts eaten is accompanied by an increase in the severity
of the disease.
- Strain of coccidia. Different strains of a species may vary in pathogenicity.
- Environmental factors affecting the survival of the oocysts.
- Site of development within the host. Coccidia that develop superficially are less pathogenic than those that develop deeper.
- Age of the bird. Young birds are generally more susceptible than older ones.
- Nutritional status of the host. Poorly fed birds are more susceptible.
Coccidiosis in chickens is generally classified as either intestinal or cecal. Most serious cases of intestinal coccidiosis in Alberta are caused by
E. necatrix. Cecal coccidiosis is due to E. tenella. Coccidiosis occurs most frequently in young birds. Old birds are generally immune as a result
of prior infection. Severe damage to the ceca and small intestine accompany the development of the coccidia. Broilers and layers are more
commonly infected, but broiler breeders and turkey and pheasant poults are also affected. Coccidiosis generally occurs more frequently during
warmer (May to September) than colder months (October to April) of the year. E. acervulina and E. maxima develop in epithelial cells within the
small intestine and generally cause chronic intestinal coccidiosis.
How will I know when my chickens are infected?
The most easily recognized clinical sign of severe cecal coccidiosis is the presence of bloody droppings. Dehydration may accompany cecal
coccidiosis. Coccidiosis caused by E. tenella first becomes noticeable at about three days after infection. Chickens droop, stop feeding, huddle
together and by the fourth day blood begins to appear in the droppings. The greatest amount of blood appears by day five or six and by the
eighth or ninth day the bird is either dead or on the way to recovery. Mortality is highest between the fourth and sixth days. Death may occur
unexpectedly, owing to excessive blood loss. Birds that recover may develop a chronic illness as a result of a persistent cecal core. However,
the core usually detaches itself by eight to ten days and is shed in the droppings.
E. necatrix causes a more chronic disease than E. tenella and does not produce as many oocysts. Therefore, a longer time is usually required
for high levels of environmental contamination. Birds heavily infected with E. necatrix may die before any marked change is noticed in weight or
before blood is found in the feces.
E. acervulina is less pathogenic than E. tenella or E. necatrix. E. acervulina is responsible for subacute or chronic intestinal coccidiosis in
broilers, older birds and chickens at the point of lay. The clinical signs consist of weight loss and a watery, whitish diarrhea. At postmortem,
greyish-white, pin-point foci or transversely elongated areas are visible from the outer (or serous) surface of the upper intestine. The foci
consist of dense areas of oocysts and gamete (male and female sex cells) production.
E. maxima produces few marked changes in the small intestine until the fifth day after infection. After which, in severe infections, numerous
small hemorrhages occur along with a marked production of thick mucus. The intestine loses tine and becomes flaccid and dilated. The inner
surface is inflamed and the intestinal content consists of a pinkish mucoid secretion. How can I prevent my chickens from getting coccidiosis?
A few good management practices are listed that will help control coccidiosis. Contact your veterinarian for full details.
- Anitcoccidial drugs mixed in the feed are used to limit high levels of infection.
- Keep chicks, feed and water away from droppings.
- Roost birds over wire netting if brooding arrangements make this possible.
- Place water vessels on wire frames to eliminate a concentration of wet droppings, in which the chicks can walk to pick up or spread the
- Keep litter dry and stirred frequently. Remove wet spots and replace with dry litter.
- Avoid overcrowding.
- If coccidiosis does break out, start treatment immediately.
How can I be sure my chickens have coccidia?
Diagnosis is best accomplished by postmortem examination of a representative number of birds from the flock. The location of the major lesions
gives a good indication of the species of coccidia concerned. For example, hemorrhagic lesions in the central part of the small intestine suggest
E. necatrix, those in the cecum, E. tenella. Microscopic examination of the affected areas along with measuring oocysts will confirm the
How do I treat infected birds?
Losses to the flock can be minimized by prompt chemotherapeutic treatment. Amprolium or one of the sulpha-based drugs is usually
recommended. If sulfas are used, overdosing may lead to toxicity. The emergence of drug-resistant strains of coccidia may present a major
problem. Methods used to avoid the development of drug resistance include switching classes of drugs and the "shuttle program" which is a
planned switch of drug in the middle of the bird's growing period.
Murray J. Kennedy, Ph.D. and J.A. Hanson, B.Sc., D.V.M.
Animal Health Division
|We vaccinate all day old chicks for coccidiosis with CocciVac-D2. However, coccidia vaccines do not prevent coccidiosis. It
helps build immunities to the disease by administering a small does of the live coccidia to the newly hatched chicks via spray
Some chicks can show signs of coccidia at approximately 10-14 days after vaccination. It is advised by the company not to
treat unless it is believed you are going to lose a chick.
Re - vaccination may be necessary in some environments. Sanitation and preventative measures are still necessary even
Rare and Heritage poultry